Ischemia/Reperfusion-Induced Translocation of PKCβII to Mitochondria as an Important Mediator of a Protective Signaling Mechanism in an Ischemia-Resistant Region of the Hippocampus-Reference-Cited by-同舟云学术

Ischemia/Reperfusion-Induced Translocation of PKCβII to Mitochondria as an Important Mediator of a Protective Signaling Mechanism in an Ischemia-Resistant Region of the Hippocampus

Published:2017-04-12 Issue:8 Volume:42 Page:2392-2403
ISSN:0364-3190
Container-title:Neurochemical Research
language:en
Short-container-title:Neurochem Res

Author:

Krupska Olga,Sarnowska Anna,Fedorczyk Bartlomiej,Gewartowska Magdalena,Misicka Aleksandra,Zablocka Barbara,Beresewicz Malgorzata^ORCID

Funder

Narodowe Centrum Nauki

Narodowym Centrum Nauki

KNOW

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,General Medicine,Biochemistry

Link

http://link.springer.com/article/10.1007/s11064-017-2263-3/fulltext.html

Reference43 articles.

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3. Sun X, Budas GR, Xu L, Barreto GE, Mochly-Rosen D, Giffard RG (2013) Selective activation of protein kinase C in mitochondria is neuroprotective in vitro and reduces focal ischemic brain injury in mice. J Neurosci Res 91:799–807

4. Budas GR, Churchill EN, Disatnik MH, Sun L, Mochly-Rosen D (2010) Mitochondrial import of PKCepsilon is mediated by HSP90: a role in cardioprotection from ischaemia and reperfusion injury. Cardiovasc Res 88:83–92

5. Raval AP, Dave KR, Defazio RA, Perez-Pinzon MA (2007) varepsilonPKC phosphorylates the mitochondrial K(+)(ATP) channel during induction of ischemic preconditioning in the rat hippocampus. Brain Res 1184:345–353

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