Selective activation of protein kinase C∊ in mitochondria is neuroprotective in vitro and reduces focal ischemic brain injury in mice
Author:
Affiliation:
1. Department of Anesthesia; Stanford University School of Medicine; Stanford; California
2. Department of Chemical and Systems Biology; Stanford University School of Medicine; Stanford; California
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Reference41 articles.
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2. The molecular composition of the mitochondrial permeability transition pore;Baines;J Mol Cell Cardiol,2009
3. Role of the mitochondrion in programmed necrosis;Baines;Front Physiol,2010
4. Protein kinase Cepsilon interacts with and inhibits the permeability transition pore in cardiac mitochondria;Baines;Circ Res,2003
5. Protein kinase C epsilon activates lens mitochondrial cytochrome c oxidase subunit IV during hypoxia;Barnett;Exp Eye Res,2008
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