CaMKII versus DAPK1 Binding to GluN2B in Ischemic Neuronal Cell Death after Resuscitation from Cardiac Arrest-Reference-Cited by-同舟云学术

CaMKII versus DAPK1 Binding to GluN2B in Ischemic Neuronal Cell Death after Resuscitation from Cardiac Arrest

Published:2020-01 Issue:1 Volume:30 Page:1-8.e4
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Buonarati Olivia R.,Cook Sarah G.,Goodell Dayton J.,Chalmers Nicholas E.,Rumian Nicole L.,Tullis Jonathan E.,Restrepo Susana,Coultrap Steven J.,Quillinan Nidia,Herson Paco S.,Bayer K. Ulrich

Funder

NIH

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference48 articles.

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3. Autonomous CaMKII requires further stimulation by Ca2+/calmodulin for enhancing synaptic strength;Barcomb;FASEB J.,2014

4. The CaMKII/GluN2B protein interaction maintains synaptic strength;Barcomb;J. Biol. Chem.,2016

5. NMDA receptor subunit composition controls synaptic plasticity by regulating binding to CaMKII;Barria;Neuron,2005

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