Autonomous CaMKII requires further stimulation by Ca 2+ /calmodulin for enhancing synaptic strength
Author:
Affiliation:
1. Department of PharmacologyUniversity of Colorado School of Medicine, Anschutz Medical CampusAuroraColoradoUSA
2. Department of Pediatrics, Section of NeurologyUniversity of Colorado School of Medicine, Anschutz Medical CampusAuroraColoradoUSA
Funder
National Institutes of Health
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.14-250407
Reference44 articles.
1. CaMKII regulation in information processing and storage
2. Autophosphorylation at Thr 286 of the α Calcium-Calmodulin Kinase II in LTP and Learning
3. Autonomous CaMKII Mediates Both LTP and LTD Using a Mechanism for Differential Substrate Site Selection
4. Regulation of brain Type II Ca2+/calmodulin-dependent protein kinase by autophosphorylation: A Ca2+-triggered molecular switch
5. Activation of the multifunctional Ca2+/calmodulin-dependent protein kinase by autophosphorylation: ATP modulates production of an autonomous enzyme.
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