The Mre11-Nbs1 Interface Is Essential for Viability and Tumor Suppression-Reference-Cited by-同舟云学术

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The Mre11-Nbs1 Interface Is Essential for Viability and Tumor Suppression

Published:2017-01 Issue:2 Volume:18 Page:496-507
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Kim Jun Hyun,Grosbart Malgorzata,Anand Roopesh,Wyman Claire,Cejka Petr,Petrini John H.J.

Funder

Geoffrey Beene Center at MSK Cancer Center

NIH

MSK Cancer Center

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference61 articles.

1. Rad50 is dispensable for the maintenance and viability of postmitotic tissues;Adelman;Mol. Cell. Biol.,2009

2. Synthetic lethality in ATM-deficient RAD50-mutant tumors underlies outlier response to cancer therapy;Al-Ahmadie;Cancer Discov.,2014

3. Mdm2 binds to Nbs1 at sites of DNA damage and regulates double strand break repair;Alt;J. Biol. Chem.,2005

4. Phosphorylated CtIP functions as a co-factor of the MRE11-RAD50-NBS1 endonuclease in DNA end resection;Anand;Mol. Cell,2016

5. DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation;Bakkenist;Nature,2003

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1. A rewiring of DNA replication mediated by MRE11 exonuclease underlies primed-to-naive cell de-differentiation;Cell Reports;2024-04

2. Xrs2/NBS1 promote end-bridging activity of the MRE11-RAD50 complex;Biochemical and Biophysical Research Communications;2024-02

3. The effect of NBS1 heterozygous inactivating mutation in a model of 7,12-dimethylbenz[a]anthracene-induced carcinogenesis in mice;Russian Journal of Oncology;2023-12-20

4. Importance of Germline and Somatic Alterations in Human MRE11, RAD50, and NBN Genes Coding for MRN Complex;International Journal of Molecular Sciences;2023-03-15

5. Mre11–Rad50: the DNA end game;Biochemical Society Transactions;2023-03-09

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