FXR Inhibits Endoplasmic Reticulum Stress-Induced NLRP3 Inflammasome in Hepatocytes and Ameliorates Liver Injury-Reference-Cited by-同舟云学术

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FXR Inhibits Endoplasmic Reticulum Stress-Induced NLRP3 Inflammasome in Hepatocytes and Ameliorates Liver Injury

Published:2018-09 Issue:11 Volume:24 Page:2985-2999
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Han Chang Yeob,Rho Hyun Soo,Kim Ayoung,Kim Tae Hyun,Jang Kiseok,Jun Dae Won,Kim Jong Won,Kim Bumseok,Kim Sang Geon

Funder

National Research Foundation of Korea (NRF)

Ministry of Science and ICT [MSIP]

NRF

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference43 articles.

1. NLRP3 inflammasome: from a danger signal sensor to a regulatory node of oxidative stress and inflammatory diseases;Abderrazak;Redox Biol.,2015

2. Farnesoid X receptor targeting to treat nonalcoholic steatohepatitis;Adorini;Drug Discov. Today,2012

3. Expression of liver X receptor correlates with intrahepatic inflammation and fibrosis in patients with nonalcoholic fatty liver disease;Ahn;Dig. Dis. Sci.,2014

4. New paradigms in the treatment of hepatic cholestasis: from UDCA to FXR, PXR and beyond;Beuers;J. Hepatol.,2015

5. miR-186 downregulation correlates with poor survival in lung adenocarcinoma, where it interferes with cell-cycle regulation;Cai;Cancer Res.,2013

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