Stress Kinase GCN2 Controls the Proliferative Fitness and Trafficking of Cytotoxic T Cells Independent of Environmental Amino Acid Sensing-Reference-Cited by-同舟云学术

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Stress Kinase GCN2 Controls the Proliferative Fitness and Trafficking of Cytotoxic T Cells Independent of Environmental Amino Acid Sensing

Published:2016-11 Issue:9 Volume:17 Page:2247-2258
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Van de Velde Lee-Ann,Guo Xi-Zhi J.,Barbaric Lidija,Smith Amber M.,Oguin Thomas H.,Thomas Paul G.,Murray Peter J.

Funder

NIH

Cancer Center Core

American Lebanese Syrian Associated Charities

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference33 articles.

1. GCN2 contributes to mTORC1 inhibition by leucine deprivation through an ATF4 independent mechanism;Averous;Sci. Rep.,2016

2. Tryptophan-catabolizing enzymes - party of three;Ball;Front. Immunol.,2014

3. Aryl hydrocarbon receptor control of a disease tolerance defence pathway;Bessede;Nature,2014

4. Infectious tolerance via the consumption of essential amino acids and mTOR signaling;Cobbold;Proc. Natl. Acad. Sci. USA,2009

5. Translational control of hippocampal synaptic plasticity and memory by the eIF2alpha kinase GCN2;Costa-Mattioli;Nature,2005

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3. The aryl hydrocarbon receptor and FOS mediate cytotoxicity induced by Acinetobacter baumannii;Nature Communications;2024-09-11

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