Drug-induced eRF1 degradation promotes readthrough and reveals a new branch of ribosome quality control-Reference-Cited by-同舟云学术

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Drug-induced eRF1 degradation promotes readthrough and reveals a new branch of ribosome quality control

Published:2023-09 Issue:9 Volume:42 Page:113056
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Gurzeler Lukas-Adrian,Link Marion,Ibig Yvonne,Schmidt Isabel,Galuba Olaf,Schoenbett Julian,Gasser-Didierlaurant Christelle,Parker Christian N.,Mao Xiaohong,Bitsch Francis,Schirle Markus,Couttet Philipp,Sigoillot Frederic,Ziegelmüller Jana,Uldry Anne-Christine,Teodorowicz Wojciech,Schmiedeberg Niko,Mühlemann Oliver,Reinhardt Jürgen

Funder

Kanton Bern

Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung

nccr – on the move

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference80 articles.

1. A meta-analysis of nonsense mutations causing human genetic disease;Mort;Hum. Mutat.,2008

2. Stop-codon read-through for patients affected by a lysosomal storage disorder;Brooks;Trends Mol. Med.,2006

3. Advances in therapeutic use of a drug-stimulated translational readthrough of premature termination codons;Dabrowski;Mol. Med.,2018

4. Pharmaceutical therapies to recode nonsense mutations in inherited diseases;Lee;Pharmacol. Ther.,2012

5. Targeting Nonsense Mutations in Diseases with Translational Read-Through-Inducing Drugs (TRIDs);Nagel-Wolfrum;BioDrugs,2016

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1. The integrated stress response regulates 18S nonfunctional rRNA decay in mammals;2024-07-30

2. Dysregulated ribosome quality control in human diseases;The FEBS Journal;2024-07

3. Exploring the multifaceted role of GCN1: Implications in cellular responses and disease pathogenesis;Biomedicine & Pharmacotherapy;2024-06

4. Translation velocity determines the efficacy of engineered suppressor tRNAs on pathogenic nonsense mutations;Nature Communications;2024-04-05

5. Eukaryotic release factor 1 from Euplotes promotes frameshifting at premature stop codons in human cells;iScience;2024-04

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