Advances in therapeutic use of a drug-stimulated translational readthrough of premature termination codons-Reference-Cited by-同舟云学术

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Advances in therapeutic use of a drug-stimulated translational readthrough of premature termination codons

Published:2018-05-29 Issue:1 Volume:24 Page:
ISSN:1076-1551
Container-title:Molecular Medicine
language:en
Short-container-title:Mol Med

Author:

Dabrowski Maciej,Bukowy-Bieryllo Zuzanna,Zietkiewicz Ewa

Funder

National Science Centre, Poland

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics,Molecular Biology,Molecular Medicine

Link

http://link.springer.com/content/pdf/10.1186/s10020-018-0024-7.pdf

Reference161 articles.

1. Abi-Hachem RN, Zine A, Van De Water TR. The injured cochlea as a target for inflammatory processes, initiation of cell death pathways and application of related otoprotectives strategies. Recent Patents CNS Drug Discov. 2010;5:147–63.

2. Allamand V, Bidou L, Arakawa M, et al. Drug-induced readthrough of premature stop codons leads to the stabilization of laminin alpha2 chain mRNA in CMD myotubes. J Gene Med. 2008;10:217–24. https://doi.org/10.1002/jgm.1140 .

3. Amrani N, Ganesan R, Kervestin S, et al. A faux 3’-UTR promotes aberrant termination and triggers nonsense-mediated mRNA decay. Nature. 2004;432:112–8. https://doi.org/10.1038/nature03060 .

4. Anderson WF, Gorini L, Breckenridge L. Role of ribosomes in streptomycin-activated suppression. Proc Natl Acad Sci U S A. 1965;54:1076–83.

5. Arakawa M, Shiozuka M, Nakayama Y, et al. Negamycin restores dystrophin expression in skeletal and cardiac muscles of mdx mice. J Biochem (Tokyo). 2003;134:751–8.

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