Histone deacetylase 3 contributes to the antiviral innate immunity of macrophages by interacting with FOXK1 to regulate STAT1/2 transcription-Reference-Cited by-同舟云学术

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Histone deacetylase 3 contributes to the antiviral innate immunity of macrophages by interacting with FOXK1 to regulate STAT1/2 transcription

Published:2022-01 Issue:4 Volume:38 Page:110302
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Yang Liping,Chen Shengchuan,Zhao Qun,Pan Chaohu,Peng Linan,Han Yu,Li Lili,Ruan Jiayin,Xia Jingyan,Yang Heng,Xu Feng,Cheng Genhong

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference51 articles.

1. STAT1 signaling is not regulated by a phosphorylation-acetylation switch;Antunes;Mol. Cell Biol,2011

2. STAT2 nuclear trafficking;Banninger;J. Biol. Chem.,2004

3. MitoQ protects against hyperpermeability of endothelium barrier in acute lung injury via a Nrf2-dependent mechanism;Cen;Redox Biol.,2021

4. Induction of interferon-stimulated gene expression and antiviral responses require protein deacetylase activity;Chang;Proc. Natl. Acad. Sci. U S A.,2004

5. Requirement for the histone deacetylase Hdac3 for the inflammatory gene expression program in macrophages;Chen;Proc. Natl. Acad. Sci. U S A.,2012

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