Identification of a Post-translational Modification with Ribitol-Phosphate and Its Defect in Muscular Dystrophy-Reference-Cited by-同舟云学术

Identification of a Post-translational Modification with Ribitol-Phosphate and Its Defect in Muscular Dystrophy

Published:2016-03 Issue:9 Volume:14 Page:2209-2223
ISSN:2211-1247
Container-title:Cell Reports
language:en
Short-container-title:Cell Reports

Author:

Kanagawa Motoi,Kobayashi Kazuhiro,Tajiri Michiko,Manya Hiroshi,Kuga Atsushi,Yamaguchi Yoshiki,Akasaka-Manya Keiko,Furukawa Jun-ichi,Mizuno Mamoru,Kawakami Hiroko,Shinohara Yasuro,Wada Yoshinao,Endo Tamao,Toda Tatsushi

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference44 articles.

1. Reduced expression of fukutin related protein in mice results in a model for fukutin related protein associated muscular dystrophies;Ackroyd;Brain,2009

2. The fukutin protein family--predicted enzymes modifying cell-surface molecules;Aravind;Curr. Biol.,1999

3. Tumor suppressor function of laminin-binding α-dystroglycan requires a distinct β3-N-acetylglucosaminyltransferase;Bao;Proc. Natl. Acad. Sci. USA,2009

4. Synthesis of CDP-activated ribitol for teichoic acid precursors in Streptococcus pneumoniae;Baur;J. Bacteriol.,2009

5. Mutations in the fukutin-related protein gene (FKRP) cause a form of congenital muscular dystrophy with secondary laminin α2 deficiency and abnormal glycosylation of α-dystroglycan;Brockington;Am. J. Hum. Genet.,2001

Cited by 189 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Exploring domain architectures of human glycosyltransferases: Highlighting the functional diversity of non-catalytic add-on domains;Biochimica et Biophysica Acta (BBA) - General Subjects;2024-10

2. Developing Method for Minor Acidic O-Glycan Analysis in Mucin and Cancer Cell Samples;Journal of Proteome Research;2024-09-10

3. Solute Carrier Family 35 (SLC35)—An Overview and Recent Progress;Biologics;2024-08-15

4. Dual FKRP/FST gene therapy normalizes ambulation, increases strength, decreases pathology, and amplifies gene expression in LGMDR9 mice;Molecular Therapy;2024-08

5. Regulatory Mechanisms of Laminin-Binding <i>O</i>-Mannosyl Glycan Biosynthesis;Trends in Glycoscience and Glycotechnology;2024-05-25