Targeting the Shc-EGFR interaction with indomethacin inhibits MAP kinase pathway signalling-Reference-Cited by-同舟云学术

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Targeting the Shc-EGFR interaction with indomethacin inhibits MAP kinase pathway signalling

Published:2019-08 Issue: Volume:457 Page:86-97
ISSN:0304-3835
Container-title:Cancer Letters
language:en
Short-container-title:Cancer Letters

Author:

Lin Chi-Chuan,Suen Kin Man,Stainthorp Amy,Wieteska Lukasz,Biggs George S.,Leitão Andrei,Montanari Carlos A.,Ladbury John E.

Funder

CRUK

São Paulo Research Foundation (FAPESP)

University of California, San Francisco

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference52 articles.

1. Interaction with Shc prevents aberrant Erk activation in the absence of extracellular stimuli;Suen;Nat. Struct. Mol. Biol.,2013

2. Key role of Shc signaling in the transforming pathway triggered by Ret/ptc2 oncoprotein;Mercalli;Oncogene,2001

3. p66Shc--a longevity redox protein in human prostate cancer progression and metastasis: p66Shc in cancer progression and metastasis;Rajendran;Cancer Metastasis Rev.,2010

4. The Shc adaptor protein is highly phosphorylated at conserved, twin tyrosine residues (Y239/240) that mediate protein-protein interactions;van der Geer;Curr. Biol.,1996

5. Evidence for a requirement for both phospholipid and phosphotyrosine binding via the Shc phosphotyrosine-binding domain in vivo;Ravichandran;Mol. Cell. Biol.,1997

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