Premetastatic shifts of endogenous and exogenous mutational processes support consolidative therapy in EGFR-driven lung adenocarcinoma

Author:

Fisk J. NicholasORCID,Mahal Amandeep R.ORCID,Dornburg Alex,Gaffney Stephen G.ORCID,Aneja Sanjay,Contessa Joseph N.,Rimm David,Yu James B.ORCID,Townsend Jeffrey P.ORCID

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference34 articles.

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3. Heterogeneous mechanisms of primary and acquired resistance to third-generation EGFR inhibitors;Ortiz-Cuaran;Clin. Cancer Res.,2016

4. Genotype-fitness maps of EGFR-mutant lung adenocarcinoma chart the evolutionary landscape of resistance for combination therapy optimization;Bolan;Cell Syst,2020

5. Genetic determinants of EGFR-driven lung cancer growth and therapeutic response in vivo;Foggetti;Cancer Discov.,2021

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