Effect of inducible FHIT and p53 expression in the Calu-1 lung cancer cell line

Author:

Cavazzoni A.,Galetti M.,Fumarola C.,Alfieri R.R.,Roz L.,Andriani F.,Carbognani P.,Rusca M.,Sozzi G.,Petronini P.G.

Publisher

Elsevier BV

Subject

Cancer Research,Oncology

Reference26 articles.

1. The FHIT gene 3p14.2 is abnormal in lung cancer;Sozzi;Cell,1996

2. Loss of FHIT function in lung cancer and preinvasive bronchial lesions;Sozzi;Cancer Res.,1998

3. Prognostic relevance of fragile histidine triad protein expression in patients with small cell lung cancer;Rohr;Clin. Cancer Res.,2005

4. Loss of FHIT protein expression is related to high proliferation, low apoptosis and worse prognosis in non-small-cell lung cancer;Toledo;Mod. Pathol.,2004

5. FHIT a putative tumor suppressor in humans, is a dinucleoside 5′,5‴-P1,P3-triphosphate hydrolase;Barnes;Biochemistry,1996

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