Signaling Crosstalk of FHIT, p53, and p38 in etoposide‐induced apoptosis in MCF‐7 cells-Reference-Cited by-同舟云学术

Signaling Crosstalk of FHIT, p53, and p38 in etoposide‐induced apoptosis in MCF‐7 cells

Published:2019-01-04 Issue:6 Volume:120 Page:9125-9137
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J Cell Biochem

Author:

Khedri Azam¹^ORCID,Khaghani Shahnaz¹,Kheirollah Alireza²,Babaahmadi‐Rezaei Hossein³,Shadboorestan Amir⁴,Zangooei Mohammad¹,Afra Hajar Shokri¹,Meshkani Reza¹^ORCID,Ghahremani Mohammad Hossein⁴^ORCID

Affiliation:

1. Department of Biochemistry Faculty of Medicine, Tehran University of Medical Sciences Tehran Iran

2. Cellular and Molecular Research Center, Department of Clinical Biochemistry, Faculty of Medicine, Ahvaz Jundishapur University of Medical Sciences Ahvaz Iran

3. Hyperlipidemia Research Center, Department of Clinical Biochemistry Faculty of Medicine, Ahvaz Jundishapur University of Medical Sciences Ahvaz Iran

4. Department of Toxicology Pharmacology Faculty of Pharmacy, Tehran University of Medical Sciences Tehran Iran

Funder

Deputy of Research, Tehran University of Medical Sciences

NIMAD

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.28188

Reference43 articles.

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2. The FHIT Gene, Spanning the Chromosome 3p14.2 Fragile Site and Renal Carcinoma–Associated t(3;8) Breakpoint, Is Abnormal in Digestive Tract Cancers

3. Do FHIT gene alterations play a role in human solid tumors?

4. The FHIT gene product: tumor suppressor and genome “caretaker”

5. Effect of FHIT gene replacement on growth, cell cycle and apoptosis in pancreatic cancer cells

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