Induction of SOS functions in Escherichia coli by lesions resulting from incorporation of 5-bromouracil into DNA

Author:

Pietrzykowska Irena,Krych Małgorzata,Shugar David

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Genetics,Molecular Biology

Reference38 articles.

1. Inducibility of a gene product required for UV and chemical mutagenesis in Escherichia coli;Bagg,1981

2. Total substitution of bromodeoxyuridine for thymidine in DNA of a bromodeoxyuridine-dependent cell line;Bick;Proc. Natl. Acad. Sci. (U.S.A.),1974

3. 5-Bromouracil-tolerant mutants of B. subtilis;Bishop;J. Bacteriol.,1972

4. Thermal denaturation of DNA from bromodeoxyuridine-substituted cells;Cullen;Nucleic Acid Res.,1976

5. Bromodeoxyuridine dependence: A new mutation in mammalian cells;Davidson,1973

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1. Molecular mechanisms of sister-chromatid exchange;Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis;2007-03

2. A new mutation inEscherichia coli K12,isfA, which is responsible for inhibition of SOS functions;Molecular and General Genetics MGG;1995-07

3. Mechanism of lead(II) bromide mutagenesis;Toxicological & Environmental Chemistry;1990-08

4. Induction of the SOS response in Escherichia coli by azidothymidine and dideoxynucleosides;Antimicrobial Agents and Chemotherapy;1990-06

5. Ability of base analogs to induce the SOS response: effect of a dam mutation and mismatch repair system;Molecular and General Genetics MGG;1985-12

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