Selective protein arylation by acetaminophen and 2,6-dimethylacetaminophen in cultured hepatocytes from phenobarbital-induced and uninduced mice-Reference-Cited by-同舟云学术

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Selective protein arylation by acetaminophen and 2,6-dimethylacetaminophen in cultured hepatocytes from phenobarbital-induced and uninduced mice

Published:1989-12 Issue:24 Volume:38 Page:4429-4438
ISSN:0006-2952
Container-title:Biochemical Pharmacology
language:en
Short-container-title:Biochemical Pharmacology

Author:

Birge Raymond B.,Bartolone John B.,McCann Denis J.,Mangold James B.,Cohen Steven D.,Khairallah Edward A.

Publisher

Elsevier BV

Subject

Pharmacology,Biochemistry

Reference44 articles.

1. Acetaminophen-induced hepatic necrosis. IV. Protective role of glutathione;Mitchell;J Pharmacol Exp Ther,1973

2. Acetaminophen-induced hepatic necrosis. II. Role of covalent binding in vivo;Jollow;J Pharmacol Exp Ther,1973

3. Clinical features of acetaminophen toxicity;McClain;J Clin Gastroenterol,1988

4. N-Acetyl-p-benzoquinone imine: a cytochrome P-450- mediated oxidation product of acetaminophen;Dahlin,1984

5. Biochemical toxicology of acetaminophen;Hinson,1980

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1. Mitochondrial protein adducts formation and mitochondrial dysfunction during N-acetyl-m-aminophenol (AMAP)-induced hepatotoxicity in primary human hepatocytes;Toxicology and Applied Pharmacology;2015-12

2. Use of a systems model of drug-induced liver injury (DILIsym®) to elucidate the mechanistic differences between acetaminophen and its less-toxic isomer, AMAP, in mice;Toxicology Letters;2014-04

3. Mechanisms of Acetaminophen-Induced Liver Disease;Drug-Induced Liver Disease;2013

4. Dose-dependent transitions in mechanisms of toxicity: case studies;Toxicology and Applied Pharmacology;2004-12-15

5. Type 1 Diabetic Mice Are Protected from Acetaminophen Hepatotoxicity;Toxicological Sciences;2003-04-15

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