Tissue-Specific Knockout of the Insulin Receptor in Pancreatic β Cells Creates an Insulin Secretory Defect Similar to that in Type 2 Diabetes-Reference-Cited by-同舟云学术

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Tissue-Specific Knockout of the Insulin Receptor in Pancreatic β Cells Creates an Insulin Secretory Defect Similar to that in Type 2 Diabetes

Published:1999-02 Issue:3 Volume:96 Page:329-339
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Kulkarni Rohit N,Brüning Jens C,Winnay Jonathon N,Postic Catherine,Magnuson Mark A,Kahn C.Ronald

Funder

Deutsche Forschungsgemeinschaft

National Institutes of Health

Greenwall Foundation

Diabetes Research Center

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

Reference66 articles.

1. Early neonatal death in mice homozygous for a null allele of the insulin receptor gene;Accili;Nat. Genet.,1996

2. Indirect evidence for short-loop negative feedback of insulin secretion in the rat;Ammon;J. Endocrinol.,1991

3. Alternative pathway of insulin signaling in mice with targeted disruption of the IRS-1 gene;Araki;Nature,1994

4. Insulin suppresses its own secretion in vivo;Argoud;Diabetes,1987

5. Relationships between insulin secretion, insulin action, and fasting plasma glucose concentration in nondiabetic and non-insulin-dependent diabetic subjects;Bogardus;J. Clin. Invest.,1984

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