Early neonatal death in mice homozygous for a null allele of the insulin receptor gene-Reference-Cited by-同舟云学术

Early neonatal death in mice homozygous for a null allele of the insulin receptor gene

Published:1996-01 Issue:1 Volume:12 Page:106-109
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Accili Domenico,Drago John,Lee Eric J.,Johnson Mark D.,Cool Martha H.,Salvatore Paola,Asico Laureano D.,José Pedro A.,Taylor Simeon I.,Westphal Heiner

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng0196-106.pdf

Reference29 articles.

1. Kahn, C.R., Insulin action, diabetogenes, and the cause of type II diabetes. Diabetes 43, 1066–1084 (1994).

2. Taylor, S.I., Lilly Lecture: molecular mechanisms of insulin resistance. Lessons from patients with mutations in the insulin receptor gene. Diabetes 41, 1473–1490 (1992).

3. Accili, D. Molecular defects of the insulin receptor gene. Diab. Metab. Reviews. 1, 47–62 (1995).

4. Tamemoto, H. et al. Insulin resistance and growth retardation in mice lacking insulin receptor substrate-1. Nature 372, 182–186 (1994).

5. Araki, E. et al. Alternative pathway of insulin signalling in mice with targeted disruption of the IRS-1 gene. Nature 372, 186–190 (1994).

Cited by 521 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Distinct, common and synergistic effects of insulin and IGF-1 receptors on healthy murine ageing;Heliyon;2024-08

2. Progress in experimental models to investigate the in vivo and in vitro antidiabetic activity of drugs;Animal Models and Experimental Medicine;2024-06

3. Imprinted Grb10, encoding growth factor receptor bound protein 10, regulates fetal growth independently of the insulin-like growth factor type 1 receptor (Igf1r) and insulin receptor (Insr) genes;BMC Biology;2024-05-30

4. The insulin / IGF axis is critically important controlling gene transcription in the podocyte;2024-05-21

5. Normal Ovarian Function in Subfertile Mouse with Amhr2-Cre-Driven Ablation of Insr and Igf1r;Genes;2024-05-12