Reduced redox state allows prolonged survival of axotomized neonatal retinal ganglion cells

Author:

Geiger L.K,Kortuem K.R,Alexejun C,Levin L.A

Publisher

Elsevier BV

Subject

General Neuroscience

Reference37 articles.

1. Effects of neurotrophins on the survival and regrowth of injured retinal neurons;Aguayo;Ciba Found. Symp.,1996

2. Selective modulation of NMDA responses by reduction and oxidation;Aizenman;Neuron,1989

3. Axotomy results in delayed death and apoptosis of retinal ganglion cells in adult rats;Berkelaar;J. Neurosci.,1994

4. A critical role of the N-methyl-D-aspartate (NMDA) receptor subunit (NR) 2A in the expression of redox sensitivity of NR1/NR2A recombinant NMDA receptors;Brimecombe;J. Pharmacol. Exp. Ther.,1999

5. Axotomy-induced retinal ganglion cell death in development: its time-course and its diminution by antioxidants;Castagne;Proc. R. Soc. Lond. Biol. Sci.,1996

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