AGEs exacerbates coronary microvascular dysfunction in NoCAD by activating endoplasmic reticulum stress-mediated PERK signaling pathway-Reference-Cited by-同舟云学术

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AGEs exacerbates coronary microvascular dysfunction in NoCAD by activating endoplasmic reticulum stress-mediated PERK signaling pathway

Published:2021-04 Issue: Volume:117 Page:154710
ISSN:0026-0495
Container-title:Metabolism
language:en
Short-container-title:Metabolism

Author:

Liu Zhongwei,Zhu Haitao,Ma Yanpeng,Tang Zhiguo,Zhao Na,Wang Yuan,Pan Shuo

Funder

Northwestern Polytechnical University

Publisher

Elsevier BV

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Reference32 articles.

1. Nonobstructive coronary artery disease and risk of myocardial infarction;Maddox;JAMA,2014

2. Risk stratification of non-obstructive coronary artery disease for guidance of preventive medical therapy;Hwang;Atherosclerosis,2019

3. Coronary microvascular dysfunction: mechanisms and functional assessment;Camici;Nat Rev Cardiol,2015

4. Role of mitochondrial dysfunction in hyperglycaemia-induced coronary microvascular dysfunction: protective role of resveratrol;Joshi;Diab Vasc Dis Res,2015

5. Pathophysiology of the no-reflow phenomenon;Vrints;Acute Card Care,2009

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3. Advanced glycation end products induce skeletal muscle atrophy and insulin resistance via activating ROS-mediated ER stress PERK/FOXO1 signaling;American Journal of Physiology-Endocrinology and Metabolism;2023-03-01

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