An emerging role of deubiquitinating enzyme cylindromatosis (CYLD) in the tubulointerstitial inflammation of IgA nephropathy
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference33 articles.
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2. Treatment of IgA nephropathy;Barratt;Kidney Int.,2006
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1. Cylindromatosis lysine 63 deubiquitinase (CYLD) suppress TLR3-mediated CCL5 expression in human renal proximal tubular epithelial cells;Molecular Biology Reports;2024-09-11
2. Inhibition of Ubiquitin-specific Protease 4 Attenuates Epithelial—Mesenchymal Transition of Renal Tubular Epithelial Cells via Transforming Growth Factor Beta Receptor Type I;Current Medical Science;2022-09-30
3. Podocyte RNF166 deficiency alleviates diabetic nephropathy by mitigating mitochondria impairment and apoptosis via regulation of CYLD signal;Biochemical and Biophysical Research Communications;2021-03
4. Inhibition of USP4 attenuates epithelial-mesenchymal transition of renal tubular epithelial cells by TβRI;2020-10-28
5. Microglial activation in the dorsal striatum participates in anxiety-like behavior in Cyld knockout mice;Brain, Behavior, and Immunity;2020-10
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