Podocyte RNF166 deficiency alleviates diabetic nephropathy by mitigating mitochondria impairment and apoptosis via regulation of CYLD signal
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference25 articles.
1. Diabetic nephropathy: perspective on novel molecular mechanisms;Gnudi;Trends Endocrinol. Metabol.,2016
2. Progressive renal decline: the new paradigm of diabetic nephropathy in type 1 diabetes;Krolewski;Diabetes Care,2015
3. Podocytopathy in diabetes: a metabolic and endocrine disorder;Diez-Sampedro;Am. J. Kidney Dis.,2011
4. Swiprosin-1 promotes mitochondria-dependent apoptosis of glomerular podocytes via P38 MAPK pathway in early-stage diabetic nephropathy;Wang;Cell. Physiol. Biochem.,2018
5. Emodin mitigates podocytes apoptosis induced by endoplasmic reticulum stress through the inhibition of the PERK pathway in diabetic nephropathy;Tian;Drug Des. Dev. Ther.,2018
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1. Renal aging and mitochondrial quality control;Biogerontology;2024-02-13
2. Biofactors regulating mitochondrial function and dynamics in podocytes and podocytopathies;Journal of Cellular Physiology;2023-09-02
3. The Potential of Cylindromatosis (CYLD) as a Therapeutic Target in Oxidative Stress-Associated Pathologies: A Comprehensive Evaluation;International Journal of Molecular Sciences;2023-05-06
4. A natural products solution to diabetic nephropathy therapy;Pharmacology & Therapeutics;2023-01
5. Intermittent Fasting—A Healthy Dietary Pattern for Diabetic Nephropathy;Nutrients;2022-09-26
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