Hyaluronic acid and its receptor CD44, acting through TMEM2, inhibit morphological differentiation in oligodendroglial cells
Author:
Funder
Ministry of Education, Culture, Sports, Science and Technology
Ministry of Health, Labour and Welfare
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference37 articles.
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3. Coaxial electrospun PLLA fibers modified with water-soluble materials for oligodendrocyte myelination;Liu;Polymers,2021
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5. Hyaluronan as an immune regulator in human diseases;Jiang;Physiol. Rev.,2011
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1. Hyaluronic acid as a tumor progression agent and a potential chemotherapeutic biomolecule against cancer: A review on its dual role;International Journal of Biological Macromolecules;2024-08
2. Naked Mole-Rat Tmem2 Lacks Physiological Hyaluronan-Degrading Activity and Resembles Human Tmem2 More Closely than Active Mouse Tmem2;2024
3. Human TMEM2 is not a catalytic hyaluronidase, but a regulator of hyaluronan metabolism via HYBID (KIAA1199/CEMIP) and HAS2 expression;Journal of Biological Chemistry;2023-06
4. Hypomyelinating Leukodystrophy 10 (HLD10)-Associated Mutations of PYCR2 Form Large Size Mitochondria, Inhibiting Oligodendroglial Cell Morphological Differentiation;Neurology International;2022-12-16
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