Inhibition of CX3CR1 reduces cell motility and viability in pancreatic adenocarcinoma epithelial cells-Reference-Cited by-同舟云学术

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Inhibition of CX3CR1 reduces cell motility and viability in pancreatic adenocarcinoma epithelial cells

Published:2018-01 Issue:3 Volume:495 Page:2264-2269
ISSN:0006-291X
Container-title:Biochemical and Biophysical Research Communications
language:en
Short-container-title:Biochemical and Biophysical Research Communications

Author:

Stout Matthew C.,Narayan Shilpa,Pillet Emily S.,Salvino Joseph M.^ORCID,Campbell Paul M.

Funder

PhRMA Foundation

W.W. Smith Charitable Trust

Drexel University's Clinical & Translational Research Institute

American Cancer Society

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference42 articles.

1. Cancer statistics, 2017;Siegel;CA Canc. J. Clin.,2017

2. Nanomedicine strategies to overcome the pathophysiological barriers of pancreatic cancer;Adiseshaiah;Nat. Rev. Clin. Oncol.,2016

3. KRAS: feeding pancreatic cancer proliferation;Bryant;Trends Biochem. Sci.,2014

4. Disruption of p16 and activation of Kras in pancreas increase ductal adenocarcinoma formation and metastasis in vivo;Qiu;Oncotarget,2011

5. Depletion of carcinoma-associated fibroblasts and fibrosis induces immunosuppression and accelerates pancreas cancer with reduced survival;Ozdemir;Canc. Cell,2014

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2. Molecular pathway of pancreatic cancer‐associated neuropathic pain;Journal of Biochemical and Molecular Toxicology;2024-04

3. Fractalkine Signalling (CX3CL1/CX3CR1 Axis) as an Emerging Target in Coronary Artery Disease;Journal of Clinical Medicine;2023-07-21

4. Nucleus Pulposus Cells Induce M2 Polarization of RAW264.7 via CX3CL1/CX3CR1 Pathway and M2 Macrophages Promote Proliferation and Anabolism of Nucleus Pulposus Cells;Stem Cells International;2023-02-20

5. Structure and Function of Ligand CX3CL1 and its Receptor CX3CR1 in Cancer;Current Medicinal Chemistry;2022-12

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