Oltipraz-induced phase 2 enzyme response conserved in cells lacking mitochondrial DNA

Author:

Chua Yee Liu,Zhang Dawei,Boelsterli Urs,Moore Philip K.,Whiteman Matthew,Armstrong Jeffrey S.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Biochemistry,Biophysics

Reference28 articles.

1. Mechanism of protection against aflatoxin tumorigenicity in rats fed 5-(2-pyrazinyl)-4-methyl-1, 2-dithiol-3-thione (oltipraz) and related 1, 2-dithiol-3-thiones and 1, 2-dithiol-3-ones;Kensler;Cancer Res.,1987

2. Transcriptional control of glutathione S-transferase gene expression by the chemopreventive agent of 5-(2-pyrazinyl)-4-methyl-1, 2-dithiol-3-thione (oltipraz) in rat liver;Davidson;Cancer Res.,1990

3. Isolation of cDNAs representing dithiolthione-responsive genes;Primiano;Carcinogenesis,1996

4. A comparison of the effect of inducers on the expression of glutathione-S-transferases in the liver of the intact rat and in hepatocytes in primary culture;Langouet;Hepatology,1996

5. Protection of retinal pigment epithelial cells from oxidative damage by oltipraz, a cancer chemopreventive agent;Nelson;Invest. Ophthalmol. Vis. Sci.,2002

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