Is Antioxidant Potential of the Mitochondrial Targeted Ubiquinone Derivative MitoQ Conserved in Cells Lacking mtDNA?-Reference-Cited by-同舟云学术

Is Antioxidant Potential of the Mitochondrial Targeted Ubiquinone Derivative MitoQ Conserved in Cells Lacking mtDNA?

Published:2008-03 Issue:3 Volume:10 Page:651-660
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Lu Chao¹,Zhang Dawei¹,Whiteman Matthew²,Armstrong Jeffrey S.¹

Affiliation:

1. Department of Biochemistry, Yong Loo Lin School of Medicine, National University of Singapore, Republic of Singapore.

2. Peninsula Medical School, Universities of Exeter and Plymouth, St Luke's Campus, Exeter, England.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2007.1865

Reference28 articles.

1. Targeting an antioxidant to mitochondria decreases cardiac ischemia‐reperfusion injury

2. Sequence and organization of the human mitochondrial genome

3. Mitochondrial membrane permeabilization: the sine qua non for cell death

4. Glutathione depletion enforces the mitochondrial permeability transition and causes cell death in HL60 cells that overexpress Bcl‐2

5. The Coenzyme Q10 Analog Decylubiquinone Inhibits the Redox-activated Mitochondrial Permeability Transition

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