Dock5 controls the peripheral B cell differentiation via regulating BCR signaling and actin reorganization
Author:
Funder
Natural Science Foundation of China
Publisher
Elsevier BV
Subject
Immunology
Reference27 articles.
1. Allosteric inhibition of the guanine nucleotide exchange factor DOCK5 by a small molecule;Ferrandez;Sci. Rep.,2017
2. DOCK2 and DOCK5 act additively in neutrophils to regulate chemotaxis, superoxide production, and extracellular trap formation;Watanabe;J. Immunol.,2014
3. Immune regulatory functions of DOCK family proteins in health and disease;Nishikimi;Exp. Cell Res.,2013
4. Upregulation of multiple signaling pathways by Dock5 deletion in epithelial cells;Xu;Mol. Vis.,2017
5. Pharmacological inhibition of Dock5 prevents osteolysis by affecting osteoclast podosome organization while preserving bone formation;Vives;Nat. Commun.,2015
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1. Dock5 Deficiency Promotes Proteinuric Kidney Diseases via Modulating Podocyte Lipid Metabolism;Advanced Science;2023-12-31
2. Exome hits demystified: The next frontier;Asian Journal of Psychiatry;2021-05
3. The regulation of DOCK family proteins on T and B cells;Journal of Leukocyte Biology;2020-06-15
4. Corrigendum to “Dock5 controls the peripheral B cell differentiation via regulating BCR signaling and actin reorganization” [Cell. Immunol. 337 (2019) 15–21];Cellular Immunology;2020-05
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