Activation of AMPK Attenuated Cardiac Fibrosis by Inhibiting CDK2 via p21/p27 and miR-29 Family Pathways in Rats-Reference-Cited by-同舟云学术

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Activation of AMPK Attenuated Cardiac Fibrosis by Inhibiting CDK2 via p21/p27 and miR-29 Family Pathways in Rats

Published:2017-09 Issue: Volume:8 Page:277-290
ISSN:2162-2531
Container-title:Molecular Therapy - Nucleic Acids
language:en
Short-container-title:Molecular Therapy - Nucleic Acids

Author:

Qi Hanping,Liu Yan,Li Shuzhi,Chen Yunping,Li Lei,Cao Yonggang,E Mingyao,Shi Pilong,Song Chao,Li Baiyan,Sun Hongli

Funder

Yu Weihan Outstanding Youth Training Fund

Harbin Medical University

Natural Science Foundation

Heilongjiang Province

Wu Liande Fund

Seed Fund

Project of Science and Technology Bureau of Daqing

Publisher

Elsevier BV

Subject

Drug Discovery,Molecular Medicine

Reference31 articles.

1. The origin of fibroblasts and mechanism of cardiac fibrosis;Krenning;J. Cell. Physiol.,2010

2. Expression and accumulation of interstitial collagen in the neonatal rat heart;Carver;Anat. Rec.,1993

3. AMP-activated protein kinase--development of the energy sensor concept;Hardie;J. Physiol.,2006

4. High rates of fatty acid oxidation during reperfusion of ischemic hearts are associated with a decrease in malonyl-CoA levels due to an increase in 5′-AMP-activated protein kinase inhibition of acetyl-CoA carboxylase;Kudo;J. Biol. Chem.,1995

5. Characterization of 5'AMP-activated protein kinase activity in the heart and its role in inhibiting acetyl-CoA carboxylase during reperfusion following ischemia;Kudo;Biochim. Biophys. Acta,1996

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