PPARγ activators down-regulate the expression of PPARγ in 3T3-L1 adipocytes
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(99)00268-9/fullpdf
Reference27 articles.
1. Development of hormone receptors and hormonal responsiveness in vitro. Insulin receptors and insulin sensitivity in the preadipocyte and adipocyte forms of 3T3-L1 cells.
2. A direct role for C/EBP and the AP-I-binding site in gene expression linked to adipocyte differentiation.
3. Differentiation-induced gene expression in 3T3-L1 preadipocytes: CCAAT/enhancer binding protein interacts with and activates the promoters of two adipocyte-specific genes.
4. CCAAT-Enhancer Binding Protein: A Component of a Differentiation Switch
5. Activation of a member of the steroid hormone receptor superfamily by peroxisome proliferators
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