Stimulation of p38 MAP kinase reduces acidosis and Na+overload in preconditioned hepatocytes
Author:
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1016/S0014-5793(01)02189-5/fullpdf
Reference30 articles.
1. Protective effect of preconditioning on the injury associated to hepatic ischemia-reperfusion in the rat: Role of nitric oxide and adenosine
2. Amelioration of liver injury by ischaemic preconditioning
3. Ischemic preconditioning protects the mouse liver by inhibition of apoptosis through a caspase-dependent pathway
4. Ischemic preconditioning reduces Na+ accumulation and cell killing in isolated rat hepatocytes exposed to hypoxia
5. PROTECTIVE EFFECT OF ISCHEMIC PRECONDITIONING ON LIVER PRESERVATION-REPERFUSION INJURY IN RATS
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2. Differential consequences of protein kinase C activation during early and late hepatic ischemic preconditioning;The Journal of Physiological Sciences;2012-02-23
3. The nitric oxide pathway - evidence and mechanisms for protection against liver ischaemia reperfusion injury;Liver International;2012-02-08
4. Interleukin-1 Stimulates Glutamate Uptake in Glial Cells by Accelerating Membrane Trafficking of Na + /K + -ATPase via Actin Depolymerization;Molecular and Cellular Biology;2008-05-15
5. Role of p38 map kinase in glycine-induced hepatocyte resistance to hypoxic injury;Journal of Hepatology;2007-04
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