Corneal stromal localization of TGF beta isoforms in spontaneous persistent epithelial defects after PRK in rabbits-Reference-Cited by-同舟云学术

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Corneal stromal localization of TGF beta isoforms in spontaneous persistent epithelial defects after PRK in rabbits

Published:2024-02 Issue: Volume:239 Page:109794
ISSN:0014-4835
Container-title:Experimental Eye Research
language:en
Short-container-title:Experimental Eye Research

Author:

Villabona-Martinez Valeria,Dutra Barbara A.L.,Sampaio Lycia P.,Santhiago Marcony R.,Wilson Steven E.^ORCID

Funder

Research to Prevent Blindness

National Institutes of Health

Cleveland Eye Bank Foundation

U.S. Department of Defense

National Eye Institute

Publisher

Elsevier BV

Reference15 articles.

1. TGFβ1 and TGFβ2 proteins in corneas with and without stromal fibrosis: delayed regeneration of epithelial barrier function and the epithelial basement membrane in corneas with stromal fibrosis;de Oliveira;Exp. Eye Res.,2021

2. Epithelial basement membrane regeneration after PRK-induced epithelial-stromal injury in rabbits: fibrotic vs. non-fibrotic corneal healing;de Oliveira;J. Refract. Surg.,2022

3. Reversal of fibrosis by TGF-β3 in a 3D in vitro model;Karamichos;Exp. Eye Res.,2014

4. Transforming growth factor-β3 regulates assembly of a non-fibrotic matrix in a 3D corneal model;Karamichos;J. Tissue Eng. Regen. Med.,2011

5. Topical losartan inhibits corneal scarring fibrosis and collagen type IV deposition after Descemet's membrane-endothelial excision in rabbits;Sampaio;Exp. Eye Res.,2022

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