RAN Translation: Fragile X in the Running
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference15 articles.
1. Unconventional Translation of C9ORF72 GGGGCC Expansion Generates Insoluble Polypeptides Specific to c9FTD/ALS
2. Specific Dephosphorylation of the Lck Tyrosine Protein Kinase at Tyr-394 by the SHP-1 Protein-tyrosine Phosphatase
3. Deletion of All CGG Repeats Plus Flanking Sequences in FMR1 Does Not Abolish Gene Expression
4. RNA-Mediated Neurodegeneration Caused by the Fragile X Premutation rCGG Repeats in Drosophila
5. The tetraplex (CGG)n destabilizing proteins hnRNP A2 and CBF-A enhance the in vivo translation of fragile X premutation mRNA
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1. Development of a selective ligand for G–G mismatches of CGG repeat RNA inducing the RNA structural conversion from the G-quadruplex into a hairpin-like structure;Organic & Biomolecular Chemistry;2022
2. Understanding the Pathogenicity of Noncoding RNA Expansion-Associated Neurodegenerative Disorders;Insights into Human Neurodegeneration: Lessons Learnt from Drosophila;2019
3. Integration-independent Transgenic Huntington Disease Fragment Mouse Models Reveal Distinct Phenotypes and Life Span in Vivo;Journal of Biological Chemistry;2015-07
4. Repeat Associated Non-AUG Translation (RAN Translation) Dependent on Sequence Downstream of the ATXN2 CAG Repeat;PLOS ONE;2015-06-18
5. TMPyP4 Porphyrin Distorts RNA G-quadruplex Structures of the Disease-associated r(GGGGCC)n Repeat of the C9orf72 Gene and Blocks Interaction of RNA-binding Proteins;Journal of Biological Chemistry;2014-02
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