Response to: Pardossi-Piquard et al., “Presenilin-Dependent Transcriptional Control of the Aβ-Degrading Enzyme Neprilysin by Intracellular Domains of βAPP and APLP.” Neuron 46, 541–554
Author:
Publisher
Elsevier BV
Subject
General Neuroscience
Reference16 articles.
1. Regulation of APP-Dependent Transcription Complexes by Mint/X11s: Differential Functions of Mint Isoforms
2. Protein phosphorylation inhibits production of Alzheimer amyloid beta/A4 peptide.
3. A Transcriptively Active Complex of APP with Fe65 and Histone Acetyltransferase Tip60
4. Presenilin 1 and Presenilin 2 Have Differential Effects on the Stability and Maturation of Nicastrin in Mammalian Brain
5. Functional gamma-secretase inhibitors reduce beta-amyloid peptide levels in brain
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