Decreasing or increasing heat shock protein 72 exacerbates or attenuates heat‐induced cell death, respectively, in rat hypothalamic cells-Reference-Cited by-同舟云学术

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Decreasing or increasing heat shock protein 72 exacerbates or attenuates heat‐induced cell death, respectively, in rat hypothalamic cells

Published:2015-01 Issue:1 Volume:5 Page:724-730
ISSN:2211-5463
Container-title:FEBS Open Bio
language:en
Short-container-title:FEBS Open Bio

Author:

Lin Kao-Chang¹²,Lin Hung-Jung²³,Chang Ching-Ping²⁴,Lin Mao-Tsun²⁴

Affiliation:

1. Department of Neurology, Chi Mei Medical Center, Tainan 710, Taiwan

2. Department of Biotechnology, Tainan 710, Taiwan

3. Department of Emergency Medicine, Chi Mei Medical Center, Tainan 710, Taiwan

4. Department of Medical Research, Chi Mei Medical Center, Tainan 710, Taiwan

Funder

Ministry of Science and Technology

Chi Mei Medical Center

Publisher

Wiley

Subject

General Biochemistry, Genetics and Molecular Biology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1016/j.fob.2015.09.001

Reference29 articles.

1. Heat Shock Protein 70 (Hsp70) as an Emerging Drug Target

2. Overexpression of Rat Heat Shock Protein 70 Reduces Neuronal Injury after Transient Focal Ischemia, Transient Global Ischemia, or Kainic Acid-induced Seizures

3. Targeted hsp70.1 Disruption Increases Infarction Volume After Focal Cerebral Ischemia in Mice

4. Autoantibody response to heat shock protein 70 in patients with heatstroke

5. Decreased heat tolerance is associated with hypothalamo–pituitary–adrenocortical axis impairment

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