Overexpression of Rat Heat Shock Protein 70 Reduces Neuronal Injury after Transient Focal Ischemia, Transient Global Ischemia, or Kainic Acid-induced Seizures-Reference-Cited by-同舟云学术

Overexpression of Rat Heat Shock Protein 70 Reduces Neuronal Injury after Transient Focal Ischemia, Transient Global Ischemia, or Kainic Acid-induced Seizures

Published:2003-11 Issue:5 Volume:53 Page:1179-1188
ISSN:0148-396X
Container-title:Neurosurgery
language:en
Short-container-title:

Author:

Tsuchiya Daisuke,Hong Shwuhuey,Matsumori Yasuhiko,Kayama Takamasa,Swanson Raymond A.,Dillman Wolfgang H.,Liu Jialing,Panter S. Scott,Weinstein Philip R.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Clinical Neurology,Surgery

Link

http://academic.oup.com/neurosurgery/article-pdf/53/5/1179/17177889/00006123-200311000-00021.pdf

Reference37 articles.

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2. Overexpression of hsp70i facilitates reactivation of intracellular proteins in neurones and protects them from denaturing stress;FEBS Lett,1998

3. Heat-shock protein 70 inhibits apoptosis by preventing recruitment of procaspase-9 to the Apaf-1 apoptosome;Nat Cell Biol,2000

4. Cytosolic redistribution of cytochrome c after transient focal cerebral ischemia in rats;J Cereb Blood Flow Metab,1998

5. Overexpression of HSP72 after induction of experimental stroke protects neurons from ischemic damage;J Cereb Blood Flow Metab,2001

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