Hepatic depletion of nucleolar protein mDEF causes excessive mitochondrial copper accumulation associated with p53 and NRF1 activation-Reference-Cited by-同舟云学术

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Hepatic depletion of nucleolar protein mDEF causes excessive mitochondrial copper accumulation associated with p53 and NRF1 activation

Published:2023-07 Issue:7 Volume:26 Page:107220
ISSN:2589-0042
Container-title:iScience
language:en
Short-container-title:iScience

Author:

Wei Jinsong,Wang Shuai,Zhu Haozhe,Cui Wei,Gao Jianan,Gao Ce,Yu Bo,Liu Bojing,Chen Jun,Peng Jinrong

Funder

National Natural Science Foundation of China

National Key Research and Development Program of China

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference58 articles.

1. Wilson disease;Aggarwal;Curr. Opin. Neurol.,2020

2. The genetics of Wilson disease;Chang;Handb. Clin. Neurol.,2017

3. The Wilson disease gene is a putative copper transporting P-type ATPase similar to the Menkes gene;Bull;Nat. Genet.,1993

4. Chapter 4 - Genetic and environmental modifiers of Wilson disease;Medici,2017

5. Hepatic features of Wilson disease;Boga;Handb. Clin. Neurol.,2017

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1. Cuproptosis associated cytoskeletal destruction contributes to podocyte injury in chronic kidney disease;American Journal of Physiology-Cell Physiology;2024-08-01

2. Comprehensive analysis of the effects of the cuprotosis-associated gene SLC31A1 on patient prognosis and tumor microenvironment in human cancer;Translational Cancer Research;2024-02

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