Cuproptosis associated cytoskeletal destruction contributes to podocyte injury in chronic kidney disease-Reference-Cited by-同舟云学术

Cuproptosis associated cytoskeletal destruction contributes to podocyte injury in chronic kidney disease

Published:2024-08-01 Issue:2 Volume:327 Page:C254-C269
ISSN:0363-6143
Container-title:American Journal of Physiology-Cell Physiology
language:en
Short-container-title:American Journal of Physiology-Cell Physiology

Author:

Li Han¹²^ORCID,Fu Yingjie¹,Xu Yue¹,Miao Hongjun¹,Wang Haonan¹,Zhang Tongtong¹^ORCID,Mei Xuejian¹,He Yinglang¹,Zhang Aihua¹^ORCID,Ge Xuhua¹²^ORCID

Affiliation:

1. Department of Emergency/Critical Medicine, Children's Hospital of Nanjing Medical University, Nanjing, People’s Republic of China

2. Jiangsu Key Laboratory of Children’s Major Disease Research, Children's Hospital of Nanjing Medical University, Nanjing, People’s Republic of China

Abstract

Cuproptosis induced by excessive copper accumulation leads to podocyte cytoskeleton damage by promoting mitochondrial dysfunction, and CXCL5 acts as an upstream signal mediating the occurrence of cuproptosis.

Funder

Nanjing Medical University Science and Technology Development Fund

MOST | National Natural Science Foundation of China

Publisher

American Physiological Society

Link

https://journals.physiology.org/doi/pdf/10.1152/ajpcell.00497.2023

Reference55 articles.

1. Chronic Kidney Disease and Dietary Measures to Improve Outcomes

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5. A YAP/TAZ–ARHGAP29–RhoA Signaling Axis Regulates Podocyte Protrusions and Integrin Adhesions