Impact of mitochondrial ROS production in the pathogenesis of insulin resistance-Reference-Cited by-同舟云学术

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Impact of mitochondrial ROS production in the pathogenesis of insulin resistance

Published:2007-09 Issue:3 Volume:77 Page:S161-S164
ISSN:0168-8227
Container-title:Diabetes Research and Clinical Practice
language:en
Short-container-title:Diabetes Research and Clinical Practice

Author:

Nishikawa Takeshi,Kukidome Daisuke,Sonoda Kazuhiro,Fujisawa Kazuo,Matsuhisa Takako,Motoshima Hiroyuki,Matsumura Takeshi,Araki Eiichi

Publisher

Elsevier BV

Subject

Endocrinology,General Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine

Reference22 articles.

1. Tumor necrosis factor alpha-induced phosphorylation of insulin receptor substrate-1 (IRS-1). Possible mechanism for suppression of insulin-stimulated tyrosine phosphorylation of IRS-1;Kanety;J Biol Chem,1995

2. Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance;Hotamisligil;Science,1993

3. IRS-1-mediated inhibition of insulin receptor tyrosine kinase activity in TNF-alpha- and obesity-induced insulin resistance;Hotamisligil;Science,1996

4. Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage;Nishikawa;Nature,2000

5. Evaluation of urinary 8-hydroxydeoxy-guanosine as a novel biomarker of macrovascular complications in type 2 diabetes;Nishikawa;Diab Care,2003

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