Rapamycin restores BDNF-LTP and the persistence of long-term memory in a model of Down's syndrome-Reference-Cited by-同舟云学术

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Rapamycin restores BDNF-LTP and the persistence of long-term memory in a model of Down's syndrome

Published:2015-10 Issue: Volume:82 Page:516-525
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

Andrade-Talavera Yuniesky,Benito Itziar,Casañas Juan José,Rodríguez-Moreno Antonio,Montesinos María Luz

Funder

Ministerio de Economía y Competitividad

Fondation Jérôme Lejeune (France)

Junta de Andalucía

Publisher

Elsevier BV

Subject

Neurology

Reference49 articles.

1. Endogenous BDNF is required for long-term memory formation in the rat parietal cortex;Alonso;Learn. Mem.,2005

2. NMDA-mediated regulation of DSCAM dendritic local translation is lost in a mouse model of Down's syndrome;Alves-Sampaio;J. Neurosci.,2010

3. Presynaptic kainate receptor-mediated facilitation of glutamate release involves Ca2+ −calmodulin at mossy fiber–CA3 synapses;Andrade-Talavera;J. Neurochem.,2012

4. Persistence of long-term memory storage requires a late protein synthesis- and BDNF-dependent phase in the hippocampus;Bekinschtein;Neuron,2007

5. Excitatory–inhibitory relationship in the fascia dentata in the Ts65Dn mouse model of Down syndrome;Belichenko;J. Comp. Neurol.,2009

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