Pituitary adenylate cyclase activating polypeptide (PACAP) and its receptor 1 (PAC1) in the human infant brain and changes in the Sudden Infant Death Syndrome (SIDS)
Author:
Publisher
Elsevier BV
Subject
Neurology
Reference57 articles.
1. Impaired response to hypoxia in the respiratory center is a major cause of neonatal death of the PACAP-knockout mouse;Arata;Eur. J. Neurosci.,2013
2. Analysis of PAC1 receptor gene variants in Caucasian and African American infants dying of sudden infant death syndrome;Barrett;Acta Paediatr.,2013
3. More tryptophan hydroxylase in the brainstem dorsal raphe nucleus in depressed suicides;Boldrini;Brain Res.,2005
4. PACAP and VIP prevent apoptosis in schwannoma cells;Castorina;Brain Res.,2008
5. Effects of PACAP and VIP on hyperglycemia-induced proliferation in murine microvascular endothelial cells;Castorina;Peptides,2010
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1. Pituitary Adenylate Cyclase-Activating Polypeptide (PACAP) and Sudden Infant Death Syndrome: A Potential Model for Investigation;International Journal of Molecular Sciences;2023-10-11
2. Az adenilát-cikláz-aktiváló polipeptid (PACAP) klinikai jelentősége;Orvosi Hetilap;2023-08-20
3. Hyperthermia and Heat Stress as Risk Factors for Sudden Infant Death Syndrome: A Narrative Review;Frontiers in Pediatrics;2022-04-15
4. Morphology of the Dentate Gyrus in a Large Cohort of Sudden Infant Deaths—Interrelation Between Features but Not Diagnosis;Journal of Neuropathology & Experimental Neurology;2021-12-03
5. Genes involved in paediatric apnoea and death based on knockout animal models: Implications for sudden infant death syndrome (SIDS);Paediatric Respiratory Reviews;2021-09
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