Mitochondrial electron transport chain defects modify Parkinson's disease phenotypes in a Drosophila model-Reference-Cited by-同舟云学术

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Mitochondrial electron transport chain defects modify Parkinson's disease phenotypes in a Drosophila model

Published:2022-09 Issue: Volume:171 Page:105803
ISSN:0969-9961
Container-title:Neurobiology of Disease
language:en
Short-container-title:Neurobiology of Disease

Author:

O'Hanlon Maria E.,Tweedy Clare,Scialo Filippo,Bass Rosemary,Sanz Alberto,Smulders-Srinivasan Tora K.

Funder

European Research Council

Teesside University

Northumbria University

Publisher

Elsevier BV

Subject

Neurology

Reference85 articles.

1. A locomotor assay reveals deficits in heterozygous Parkinson’s disease model and proprioceptive mutants in adult Drosophila;Aggarwal;Proc. Natl. Acad. Sci. U. S. A.,2019

2. Assaying Locomotor, Learning, and Memory Deficits in Drosophila Models of Neurodegeneration;Ali,2011

3. High levels of mitochondrial DNA deletions in substantia nigra neurons in aging and Parkinson disease;Bender;Nat. Genet.,2006

4. Mitochondria function associated genes contribute to Parkinson’s disease risk and later age at onset;Billingsley;Npj Parkinson’s Disease.,2019

5. Respiratory chain abnormalities in skeletal muscle from patients with Parkinson’s disease;Bindoff;J. Neurol. Sci.,1991

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