Comparative analysis of the gene-inactivating potential of retroviral restriction factors APOBEC3F and APOBEC3G
Author:
Affiliation:
1. Department of Biochemistry, Microbiology and Immunology, Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada
Publisher
Microbiology Society
Subject
Virology
Reference46 articles.
1. Catalytic activity of APOBEC3F is required for efficient restriction of Vif-deficient human immunodeficiency virus
2. Different Mutagenic Potential of HIV-1 Restriction Factors APOBEC3G and APOBEC3F Is Determined by Distinct Single-Stranded DNA Scanning Mechanisms
3. APOBEC3G-Induced Hypermutation of Human Immunodeficiency Virus Type-1 Is Typically a Discrete “All or Nothing” Phenomenon
4. Possible Footprints of APOBEC3F and/or Other APOBEC3 Deaminases, but Not APOBEC3G, on HIV-1 from Patients with Acute/Early and Chronic Infections
5. Crystal Structure of Enhanced Green Fluorescent Protein to 1.35 Å Resolution Reveals Alternative Conformations for Glu222
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1. Antiretroviral APOBEC3 cytidine deaminases alter HIV-1 provirus integration site profiles;Nature Communications;2023-01-10
2. A Conserved uORF Regulates APOBEC3G Translation and Is Targeted by HIV-1 Vif Protein to Repress the Antiviral Factor;Biomedicines;2021-12-22
3. Human APOBEC3 Variations and Viral Infection;Viruses;2021-07-14
4. A conserved uORF regulates APOBEC3G translation and is targeted by HIV-1 Vif protein to repress the antiviral factor;2021-01-13
5. Mechanistic Insight into Antiretroviral Potency of 2′-Deoxy-2′-β-fluoro-4′-azidocytidine (FNC) with a Long-Lasting Effect on HIV-1 Prevention;Journal of Medicinal Chemistry;2020-07-17
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