Replication of Merkel cell polyomavirus induces reorganization of promyelocytic leukemia nuclear bodies
Author:
Affiliation:
1. Institute for Medical Microbiology, Virology and Hygiene, University Medical Center Hamburg-Eppendorf, Hamburg, Germany
2. Heinrich-Pette-Institute, Leibniz Institute for Experimental Virology, Hamburg, Germany
Publisher
Microbiology Society
Subject
Virology
Reference53 articles.
1. Epstein-Barr Virus Immediate-Early Protein BZLF1 Is SUMO-1 Modified and Disrupts Promyelocytic Leukemia Bodies
2. PML nuclear bodies and their spatial relationships in the mammalian cell nucleus
3. Regulation of apoptosis by PML and the PML-NBs
4. The Adenoviral Oncogene E1A-13S Interacts with a Specific Isoform of the Tumor Suppressor PML To Enhance Viral Transcription
5. Functional Connection between Rad51 and PML in Homology-Directed Repair
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1. The interactions between PML nuclear bodies and small and medium size DNA viruses;Virology Journal;2023-05-01
2. PML Body Component Sp100A Restricts Wild-Type Herpes Simplex Virus 1 Infection;Journal of Virology;2022-04-27
3. Dual signaling via interferon and DNA damage response elicits entrapment by giant PML nuclear bodies;eLife;2022-03-23
4. From Merkel Cell Polyomavirus Infection to Merkel Cell Carcinoma Oncogenesis;Frontiers in Microbiology;2021-09-08
5. Dual signaling via interferon and DNA damage response elicits entrapment by giant PML nuclear bodies;2021-08-26
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