PML Body Component Sp100A Restricts Wild-Type Herpes Simplex Virus 1 Infection
Author:
Affiliation:
1. Centre for Infection and Immunity Studies, School of Medicine, Shenzhen Campus of Sun Yat-sen University, Shenzhen, Guangdong, China
Abstract
Funder
National Natural Science Foundation of China
Shenzhen Science and Technology Program
Publisher
American Society for Microbiology
Subject
Virology,Insect Science,Immunology,Microbiology
Link
https://journals.asm.org/doi/pdf/10.1128/jvi.00279-22
Reference83 articles.
1. Isolation and characterization of cDNA encoding a human nuclear antigen predominantly recognized by autoantibodies from patients with primary biliary cirrhosis;Szostecki C;J Immunol,1990
2. IFN enhance expression of Sp100, an autoantigen in primary biliary cirrhosis;Guldner HH;J Immunol,1992
3. The Interferon (IFN)-stimulated Gene Sp100 Promoter Contains an IFN-γ Activation Site and an Imperfect IFN-stimulated Response Element Which Mediate Type I IFN Inducibility
4. Sequestration of PML and Sp100 Proteins in an Intranuclear Viral Structure during Herpes Simplex Virus Type 1 Infection
5. Herpes virus induced proteasome-dependent degradation of the nuclear bodies-associated PML and Sp100 proteins
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