Effects of early-life exposure to THIP on brainstem neuronal excitability in the Mecp2 -null mouse model of Rett syndrome before and after drug withdrawal
Author:
Affiliation:
1. Department of Biology; Georgia State University; Atlanta Georgia
Funder
National Institutes of Health
Publisher
Wiley
Subject
Physiology (medical),Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.14814/phy2.13110/fullpdf
Reference26 articles.
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2. Excitation/inhibition imbalance and impaired synaptic inhibition in hippocampal area CA3 of Mecp2 knockout mice;Calfa;Hippocampus,2015
3. Dysfunction in GABA signalling mediates autism-like stereotypes and Rett syndrome phenotypes;Chao;Nature,2010
4. Plasma and CNS concentrations of Gaboxadol in rats following subcutaneous administration;Cremers;Eur. J. Pharmacol.,2007
5. A mouse Mecp2-null mutation causes neurological symptoms that mimic Rett syndrome;Guy;Nat. Genet.,2001
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1. GABAergic System Dysfunction in Autism Spectrum Disorders;Frontiers in Cell and Developmental Biology;2022-02-07
2. From Cannabinoids and Neurosteroids to Statins and the Ketogenic Diet: New Therapeutic Avenues in Rett Syndrome?;Frontiers in Neuroscience;2019-07-02
3. Sexual dimorphism in brain transcriptomes of Amami spiny rats (Tokudaia osimensis): a rodent species where males lack the Y chromosome;BMC Genomics;2019-01-25
4. iPSC-derived neurons profiling reveals GABAergic circuit disruption and acetylated α-tubulin defect which improves after iHDAC6 treatment in Rett syndrome;Experimental Cell Research;2018-07
5. Effects of early-life exposure to THIP on brainstem neuronal excitability in the Mecp2 -null mouse model of Rett syndrome before and after drug withdrawal;Physiological Reports;2017-01
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