Intimal hyperplasia induced by vascular intervention causes lipoprotein retention and accelerated atherosclerosis
Author:
Affiliation:
1. Department of Molecular and Clinical Medicine; Wallenberg Laboratory; Institute of Medicine; Sahlgrenska Academy at University of Gothenburg; Gothenburg Sweden
2. Innovation Managing Direction; Center of Molecular Immunology; Havana Cuba
Funder
Vetenskapsrådet
Svenska Läkaresällskapet
Hjärt-Lungfonden
Sahlgrenska Universitetssjukhuset
Emelle foundation
Magnus Bergvalls foundation
Publisher
Wiley
Subject
Physiology (medical),Physiology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.14814/phy2.13334/fullpdf
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4. Induction of atherosclerosis in mice and hamsters without germline genetic engineering;Bjorklund;Circ. Res.,2014
5. Identification of the principal proteoglycan-binding site in LDL. A single-point mutation in apo-B100 severely affects proteoglycan interaction without affecting LDL receptor binding;Boren;J. Clin. Invest.,1998
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