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Marginal dietary zinc deprivation augments sepsis-induced alterations in skeletal muscle TNF-αbut not protein synthesis

  • Published:2016-11 Issue:21 Volume:4 Page:e13017
  • ISSN:2051-817X
  • Container-title:Physiological Reports
  • language:en
  • Short-container-title:Physiol Rep

Author:

Crowell Kristen T.1,Kelleher Shannon L.1234,Soybel David I.124,Lang Charles H.12

Affiliation:

1. Department of Surgery; College of Medicine; Pennsylvania State University; Hershey Pennsylvania

2. Department of Cellular and Molecular Physiology; College of Medicine; Pennsylvania State University; Hershey Pennsylvania

3. Department of Pharmacology; College of Medicine; Pennsylvania State University; Hershey Pennsylvania

4. Department of Nutritional Sciences; Pennsylvania State University; University Park Pennsylvania

Publisher

Wiley

Subject

Physiology (medical),Physiology

Reference60 articles.

1. Zinc status in type 2 diabetic patients: relation to the progression of diabetic nephropathy;Al-Timimi;J Clin Diagn Res,2014

2. Effect of parenteral nutrition on protein turnover in endotoxemic rats;Ash;Clin. Sci. (Lond.),1989

3. Identification of ubiquitin ligases required for skeletal muscle atrophy;Bodine;Science,2001

4. Paradoxical zinc toxicity and oxidative stress in the mammary gland during marginal dietary zinc deficiency;Bostanci;Reprod. Toxicol.,2015

5. Sustained modifications of protein metabolism in various tissues in a rat model of long-lasting sepsis;Breuille;Clin. Sci. (Lond.),1998

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