1. [7] Redwood, C. & Robinson, P. Alpha-tropomyosin mutations in inherited cardiomyopathies. J. Muscle Res. Cell Motil. 34, 285–294 (2013).
2. [12] Kawai, M. & Ishiwata, S. Use of thin filament reconstituted muscle fibres to probe the mechanism of force generation. J. Muscle Res. Cell Motil. 27, 455–468 (2006).
3. [13] Terui, T., Sodnomtseren, M., Matsuba, D., Udaka, J., Ishiwata, S., Ohtsuki, I., et al. Troponin and titin coordinately regulate length-dependent activation in skinned porcine ventricular muscle. J. Gen. Physiol. 131, 275–283 (2008).
4. [21] Bing, W., Knott, A., Redwood, C., Esposito, G., Purcell, I., Watkins, H., et al. Effect of hypertrophic cardiomyopathy mutations in human cardiac muscle alpha-tropomyosin (Asp175Asn and Glu180Gly) on the regulatory properties of human cardiac troponin determined by in vitro motility assay. J. Mol. Cell. Cardiol. 32, 1489–1498 (2000).
5. [25] Bai, F., Wang, L. & Kawai, M. A study of tropomyosin’s role in cardiac function and disease using thin-filament reconstituted myocardium. J. Muscle Res. Cell Motil. 34, 295–310 (2013).