NUP98 Rearrangement in Acute Myelomonocytic Leukemia With t(11;19)(p15;p12): The First Case Report Worldwide
Author:
Affiliation:
1. Department of Laboratory Medicine, Dong-A University College of Medicine, Busan, Korea
2. Department of Internal Medicine, Dong-A University College of Medicine, Busan, Korea
Publisher
Annals of Laboratory Medicine
Subject
Biochemistry (medical),Clinical Biochemistry,General Medicine
Link
https://synapse.koreamed.org/pdf/10.3343/alm.2017.37.3.285
Reference6 articles.
1. Nuclear Pore Component Nup98 Is a Potential Tumor Suppressor and Regulates Posttranscriptional Expression of Select p53 Target Genes
2. Proteomic analysis of the mammalian nuclear pore complex
3. Major form of NUP98/HOXC11 fusion in adult AML with t(11;12)(p15;q13) translocation exhibits aberrant trans-regulatory activity
4. The t(7;11)(p15;p15) translocation in acute myeloid leukaemia fuses the genes for nucleoporin NUP96 and class I homeoprotein HOXA9
5. Fusion of the nucleoporin gene NUP98 to HOXA9 by the chromosome translocation t(7;11)(p15;p15) in human myeloid leukaemia
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Abnormally Hypersegmented Neutrophilia in Pediatric Acute Myeloid Leukemia Associated With t(2;11)(q31;p15) and NUP98 Rearrangement;Journal of Pediatric Hematology/Oncology;2020-11-23
2. Dasatinib and navitoclax act synergistically to target NUP98-NSD1+/FLT3-ITD+ acute myeloid leukemia;Leukemia;2018-12-19
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